Bio News
Researchers at Mount Sinai in the United States identified the mechanism by which APOE4, the greatest genetic risk factor for Alzheimer’s disease, damages brain blood vessels.
APOE4 transformed pericytes, which stabilize blood vessels, into scar-forming cells, causing vascular fibrosis and amyloid accumulation.
The researchers reversed vascular damage in an aged mouse model by blocking TGF-β signaling.
Biobook Team
biobookmedia@biobook.co.krRoche’s New Drug for Uveitic Macular Edema Faces FDA Decision in July 2027
BMS Reports Concurrent Phase 3 Success in Idiopathic Pulmonary Fibrosis and Solid Tumors
Merck’s $1.3 trillion eye disease drug raises safety and efficacy concerns: “Limited competitiveness”
CHA Biotech Holds 24.23% Stake in CMG Pharmaceutical as Share Consolidation Reduces Its Share Count
Rural Development Administration Develops Two Molecular Markers to Identify Leaf Splitting in Squash
MFDS Begins Distribution of Internationally Certified Reference Materials, Including 24 New Items, for a Total of 882
