Bio News

Researchers at UT Southwestern Medical Center identified the role of SNX5, a protein that supports the immune response to tuberculosis. SNX5 was essential to the process by which immune cells present bacterial fragments on their surface to signal an infection. Mice deficient in SNX5 developed more severe lung inflammation and had a higher mortality rate.

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biobookmedia@biobook.co.krRoche’s New Drug for Uveitic Macular Edema Faces FDA Decision in July 2027
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